If this is your first visit, be sure to
check out the FAQ by clicking the
link above. You may have to register
before you can post: click the register link above to proceed. To start viewing messages,
select the forum that you want to visit from the selection below.
ma ti spari jack ogni giorno??
aahahhaha...aspetta il week end caxxuola, te ne fai mezzo litro e stai bene all week...l'alcola inibisce la capacità dell'organismo di stoccare glicogeno, aumenta la glicogenolisi (se presente)...qundi in high carb male!!
si lo so ke nn va bene,ho calcolato tutto,da sabato parte lo stop cn l'alcool,la week entrante rinizio cn alimentazione e wo. Mi sn dato alla pazza gioia giusto da martedì fine a domani sera(cercando di nn esagerare),tanto ho perso poco più di 3 kg in 4gg a causa dell'influenza..
bbeeene.,,,ora che hai perso peso è il miglior momento per allenarti tosto...la sintesi proteica sarà massima..le ipotesi sn 2:
-o sblocchi il peso
-o ti infortuni
bbeeene.,,,ora che hai perso peso è il miglior momento per allenarti tosto...la sintesi proteica sarà massima..le ipotesi sn 2:
-o sblocchi il peso
-o ti infortuni
guarda di procarioti,ribosomi e l' rna nn c'ho mai capito niente,cmq mi pare d'aver capito ke la sintesi è cosa buona ,opto per la numero 1.. lunedì parto cn la forza,x almeno 6 week
cia carissimo...yeees tutto bene,sn appena rientrato da far compere,1.5kg di bresaola x la prox settimana ke si torna a workizzare.. pagato nenake tanto 25 euri
scucuuuuuuusa...ho letto solo ora...alcuni studietti, poi ce ne sn altri ma ora ho sonno già tanto che son riuscito a rispulciare questi
Effect of alcohol intake on muscle glycogen storage after prolonged exercise
Louise M. Burke,1 Greg R. Collier,2 Elizabeth M. Broad,1 Peter G. Davis,1 David T. Martin,1 Andrew J. Sanigorski,2 and Mark Hargreaves3 1Sports Science and Sports Medicine, Australian Institute of Sport, Belconnen, Australian Capital Territory 2616; 2School of Health Sciences, Deakin University, Waurn Ponds, Victoria 3217; and 3School of Health Sciences, Deakin University, Burwood, Victoria, 3125, Australia Submitted 3 February 2003 ; accepted in final form 16 April 2003 We studied the effects of alcohol intake on postexercise muscle glycogen restoration with samples from vastus lateralis being collected immediately after glycogen-depleting cycling and after a set recovery period. Six well-trained cyclists undertook a study of 8-h recovery (2 meals), and another nine cyclists undertook a separate 24-h protocol (4 meals). In each study, subjects completed three trials in crossover order: control (C) diet [meals providing carbohydrate (CHO) of 1.75 g/kg]; alcohol-displacement (A) diet (1.5 g/kg alcohol displacing CHO energy from C) and alcohol + CHO (AC) diet (C + 1.5 g/kg alcohol). Alcohol intake reduced postmeal glycemia especially in A trial and 24-h study, although insulin responses were maintained. Alcohol intake increased serum triglycerides, particularly in the 24-h study and AC trial. Glycogen storage was decreased in A diets compared with C at 8 h (24.4 ± 7 vs. 44.6 ± 6 mmol/kg wet wt, means ± SE, P < 0.05) and 24 h (68 ± 5 vs. 82 ± 5 mmol/kg wet wt, P < 0.05). There was a trend to reduced glycogen storage with AC in 8 h (36.2 ± 8 mmol/kg wet wt, P = 0.1) but no difference in 24 h (85 ± 9 mmol/kg wet wt). We conclude that 1) the direct effect of alcohol on postexercise glycogen synthesis is unclear, and 2) the main effect of alcohol intake is indirect, by displacing CHO intake from optimal recovery nutrition practices.
Effect of ethanol on ketone metabolism André Lefèvre, Howard Adler, and Charles S. Lieber Section of Liver Disease and Nutrition, Bronx VA Hospital, New York 10468 Department of Medicine, Mt. Sinai School of Medicine of the City University of New York, New York 10468 This article has been cited by other articles in PMC. Abstract Ketonuria has been observed in alcoholics. To study the mechanism of this effect, healthy, volunteers were given adequate diets (36% of calories as lipid and 15% as protein) for 18 days, with isocaloric replacement of carbohydrate (46% of calories) by either ethanol or additional fat. The latter resulted in a high fat diet, with 82% of calories as lipid. After about 1 wk of alcohol, massive and persistent ketonuria developed. Compared with the control period, there was a 30-fold increase in fasting blood acetoacetate and β-hydroxybutyrate (P < 0.001). With the high fat diet, acetoacetate and β-hydroxybutyrate increased 8- to 10-fold (P < 0.001). In the postprandial state, ethanol also induced hyperketonemia, but less markedly than when ethanol followed an overnight fast. With low fat diets (5% of calories), alcohol (46% of total calories) did not induce ketonuria or hyperketonemia, suggesting that a combination of alcohol and dietary fat is necessary. The addition of alcohol to rat liver slices did not affect ketogenesis. In rats pretreated with alcohol for 3 days, however, ketonemia developed, hepatic glycogen was decreased, and liver slices (incubated with palmitate-14C and glucose) had a significant increase in acetoacetate production, when compared to carbohydrate pretreated controls. Alcohol pretreatment or addition of alcohol in vitro had no effect on acetoacetate utilization by rat diaphragms, and decreased only slightly the conversion of β-hydroxybutyrate-14C to 14CO2. Thus, the hyperketonemia and ketonuria observed after alcohol consumption cannot be attributed to an immediate effect of alcohol, but is the consequence of a delayed change in intermediary metabolism characterized by increased hepatic ketone production from fatty acids, possibly linked to ethanol-induced glycogen depletion and depression of citric acid cycle activity.
IL SEGUENTE INTERESSANTE PER FEMMINUCCE
The insulin-sensitizing activity of moderate alcohol consumption may promote leanness in women.McCarty MF.
Pantox Laboratories, San Diego, USA.
Cross-sectional epidemiology reveals that women who drink alcohol regularly and moderately, on average, tend to have a decidedly lower body-mass index (BMI) than non-drinking women, despite slightly higher caloric intakes. In men, moderate drinkers are no heavier than non-drinkers, yet they consume considerably more calories. The thermogenic effect which this implies is not explained by the modest acute thermic effect of ethanol ingestion. However, there is indirect evidence that regular alcohol consumption has an insulin-sensitizing effect on skeletal muscle that down-regulates insulin secretion. Decreased insulin activity on adipocytes and the liver may discourage fat storage and promote hepatic mechanisms of ketogenesis, gluconeogenesis, and associated thermogenesis, thus possibly accounting for the relative leanness of female drinkers. The possibility that prescribing moderate alcohol intake could aid weight control in non-drinking overweight females should receive clinical evaluation.
The impact of moderate drinking on risk for diabetes in women appears to be quite dramatic.
We process personal data about users of our site, through the use of cookies and other technologies, to deliver our services, personalize advertising, and to analyze site activity. We may share certain information about our users with our advertising and analytics partners. For additional details, refer to our Privacy Policy.
By clicking "I AGREE" below, you agree to our Privacy Policy and our personal data processing and cookie practices as described therein. You also acknowledge that this forum may be hosted outside your country and you consent to the collection, storage, and processing of your data in the country where this forum is hosted.
Commenta